{"id":7549,"date":"2022-11-22T13:00:22","date_gmt":"2022-11-22T10:00:22","guid":{"rendered":"https:\/\/urbancare.clinic\/fahrs-disease\/"},"modified":"2022-11-22T13:00:22","modified_gmt":"2022-11-22T10:00:22","slug":"%d0%b1%d0%be%d0%bb%d0%b5%d0%b7%d0%bd%d1%8c-%d1%84%d0%b0%d1%80%d1%81%d0%b0","status":"publish","type":"post","link":"https:\/\/urbancare.clinic\/ru\/fahrs-disease\/","title":{"rendered":"\u0411\u043e\u043b\u0435\u0437\u043d\u044c \u0424\u0430\u0440\u0441\u0430"},"content":{"rendered":"<div><img decoding=\"async\" src=\"https:\/\/urbancare.clinic\/wp-content\/uploads\/2023\/02\/fahrs-disease.jpg\" class=\"ff-og-image-inserted\" style=\"display:none\"><\/div>\n<p><span class=\"highwire-journal-article-marker-start\"><\/span><\/p>\n<p id=\"p-1\">36-\u043b\u0435\u0442\u043d\u0438\u0439 \u043c\u0443\u0436\u0447\u0438\u043d\u0430 \u043e\u0431\u0440\u0430\u0442\u0438\u043b\u0441\u044f \u0441 \u0436\u0430\u043b\u043e\u0431\u0430\u043c\u0438 \u043d\u0430 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https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>Fahrs disease - Urban Care Clinic<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/urbancare.clinic\/ru\/\u0431\u043e\u043b\u0435\u0437\u043d\u044c-\u0444\u0430\u0440\u0441\u0430\/\" \/>\n<meta property=\"og:locale\" content=\"ru_RU\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Fahrs disease - Urban Care Clinic\" \/>\n<meta property=\"og:description\" content=\"A 36-year-old man presented with a 2-month history of dizziness brought on with bending. He described the sensation of the room spinning around. The episodes were brief and lasted a few seconds. He denied any other symptoms. There was no history of trauma or other precipitating event. His neurological examination showed no motor, sensory, speech, visual field or cognitive deficit. Cranial nerves and cerebellar function were intact. There was no gaze deviation or nystagmus present. Orthostatic vital signs were negative. A CT scan of the head showed extensive bilateral symmetric intracranial calcifications involving the basal ganglia, thalamus, dentate nuclei and subcortical white matter of the cerebrum including involvement of both frontal, temporal, parietal and occipital lobes as well as the midbrain and pons (figures 1&ndash;3) consistent with Fahr&#039;s disease. A detailed discussion of the condition affecting any other family members was negative. 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