{"id":7457,"date":"2022-11-22T13:00:22","date_gmt":"2022-11-22T10:00:22","guid":{"rendered":"https:\/\/urbancare.clinic\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis\/"},"modified":"2022-11-22T13:00:22","modified_gmt":"2022-11-22T10:00:22","slug":"iperintensita-del-claustro-come-marcatore-di-encefalite-anti-nmdar","status":"publish","type":"post","link":"https:\/\/urbancare.clinic\/it\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis\/","title":{"rendered":"Iperintensit\u00e0 di Claustrum come marcatore di encefalite anti-NMDAR"},"content":{"rendered":"<div><img decoding=\"async\" src=\"https:\/\/urbancare.clinic\/wp-content\/uploads\/2023\/01\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis.jpg\" class=\"ff-og-image-inserted\" style=\"display:none\"><\/div>\n<p><span class=\"highwire-journal-article-marker-start\"><\/span><\/p>\n<p id=\"p-1\">Un giovane paziente maschio adulto presentava insorgenza subacuta, deterioramento cognitivo rapidamente progressivo e convulsioni refrattarie e gli era stata diagnosticata un&#039;encefalite definita da anticorpi anti-recettore N-metil D-aspartato (anti-NMDAR) secondo i criteri di Graus <em>et al.<\/em> (2016), in seguito al rilevamento di autoanticorpi colpevoli nel siero.<span class=\"xref-bibr\">1<\/span> La risonanza magnetica del cervello ha rivelato iperintensit\u00e0 del claustrum con immagine pesata in T2 bilaterale (T2WI) \/ recupero dell&#039;inversione attenuata dal fluido (FLAIR) (figura 1). In precedenza, in una coorte di 34 pazienti,<span class=\"xref-bibr\">2<\/span> presentandosi con epilessia refrattaria di nuova insorgenza, 4 pazienti su 34 hanno mostrato iperintensit\u00e0 FLAIR del claustrum bilaterale all&#039;imaging MRI, nessuno ... <\/p>\n<p><span class=\"highwire-journal-article-marker-end\"><\/span><a href=\"http:\/\/pmj.bmj.com\/cgi\/content\/short\/98\/1166\/e24?rss=1\">Fonte quotidiana medica<\/a><\/p>\n<p>","protected":false},"excerpt":{"rendered":"<p>Un giovane paziente maschio adulto presentava insorgenza subacuta, deterioramento cognitivo rapidamente progressivo e convulsioni refrattarie e gli era stata diagnosticata un&#039;encefalite definita da anticorpi anti-recettore N-metil D-aspartato (anti-NMDAR) secondo i criteri di Graus <i>et al.<\/i> (2016), in seguito al rilevamento di autoanticorpi colpevoli nel siero.<cross-ref type=\"bib\" refid=\"R1\">1<\/cross-ref> La risonanza magnetica del cervello ha rivelato iperintensit\u00e0 bilaterali del claustro con immagine T2 pesata (T2WI)\/recupero con inversione attenuata con fluido (FLAIR) (<cross-ref type=\"fig\" refid=\"F1\">Figura 1<\/cross-ref>). In precedenza, in una coorte di 34 pazienti,<cross-ref type=\"bib\" refid=\"R2\">2<\/cross-ref> presentandosi con epilessia refrattaria di nuova insorgenza, 4 pazienti su 34 hanno mostrato iperintensit\u00e0 FLAIR bilaterale del claustro all&#039;imaging MRI, nessuno dei quali era positivo per gli anticorpi contro gli antigeni onconeurali. Tutti questi pazienti avevano epilessie intrattabili dal punto di vista medico in cui la ripetizione dell&#039;imaging ha rivelato la risoluzione delle anomalie radiologiche, nonostante le convulsioni in corso. Il coinvolgimento radiologico della corteccia bi-insulare \u00e8 stato precedentemente segnalato nel contesto dell&#039;encefalite anti-NMDAR.<cross-ref type=\"bib\" refid=\"R3\">3<\/cross-ref> Tuttavia, nel nostro caso, non sono stati rilevati cambiamenti insulari insieme all\u2019iperintensit\u00e0 del claustro. Il paziente\u2026<\/p>","protected":false},"author":2,"featured_media":7458,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"Default","format":"standard","meta":{"page_builder":"","fifu_image_url":"https:\/\/urbancare.clinic\/wp-content\/uploads\/2023\/01\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis.jpg","fifu_image_alt":"","footnotes":""},"categories":[185],"tags":[],"class_list":["post-7457","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-research-medical-articles"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v28.4 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>Claustrum hyperintensity as a marker of anti-NMDAR encephalitis - Urban Care Clinic<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/urbancare.clinic\/it\/iperintensita-del-claustro-come-marcatore-di-encefalite-anti-nmdar\/\" \/>\n<meta property=\"og:locale\" content=\"it_IT\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Claustrum hyperintensity as a marker of anti-NMDAR encephalitis - Urban Care Clinic\" \/>\n<meta property=\"og:description\" content=\"A young adult male patient presented with subacute onset, rapidly progressive cognitive impairment and refractory seizures and was diagnosed as definite anti N-methyl D-aspartate receptor (anti-NMDAR) antibody encephalitis as per criteria by Graus et al. (2016), following detection of culprit autoantibodies in serum.1 MRI of brain revealed bilateral T2-weighted image (T2WI)\/fluid-attenuated inversion recovery (FLAIR) claustrum hyperintensities (figure 1). Previously, in a cohort of 34 patients,2 presenting with new-onset refractory epilepsy, 4 out of 34 patients showed bilateral claustrum FLAIR hyperintensities on MRI imaging, none of whom were positive for antibodies to onconeural antigens. All these patients had medically intractable epilepsies in whom repeat imaging revealed resolution of radiological abnormalities, despite ongoing seizures. Radiological involvement of bi-insular cortex has previously been reported in context of anti-NMDAR encephalitis.3 However, in our case, no insular changes were noticeable along with claustrum hyperintensities. 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