{"id":7457,"date":"2022-11-22T13:00:22","date_gmt":"2022-11-22T10:00:22","guid":{"rendered":"https:\/\/urbancare.clinic\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis\/"},"modified":"2022-11-22T13:00:22","modified_gmt":"2022-11-22T10:00:22","slug":"hyperintensite-du-claustrum-comme-marqueur-de-lencephalite-anti-nmdar","status":"publish","type":"post","link":"https:\/\/urbancare.clinic\/fr\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis\/","title":{"rendered":"Hypersignal Claustrum comme marqueur de l&#039;enc\u00e9phalite anti-NMDAR"},"content":{"rendered":"<div><img decoding=\"async\" src=\"https:\/\/urbancare.clinic\/wp-content\/uploads\/2023\/01\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis.jpg\" class=\"ff-og-image-inserted\" style=\"display:none\"><\/div>\n<p><span class=\"highwire-journal-article-marker-start\"><\/span><\/p>\n<p id=\"p-1\">Un jeune homme adulte s&#039;est pr\u00e9sent\u00e9 avec un d\u00e9but subaigu, une d\u00e9ficience cognitive rapidement progressive et des convulsions r\u00e9fractaires et a \u00e9t\u00e9 diagnostiqu\u00e9 comme une enc\u00e9phalite \u00e0 anticorps anti-r\u00e9cepteur N-m\u00e9thyl D-aspartate (anti-NMDAR) selon les crit\u00e8res de Graus <em>et coll.<\/em> (2016), suite \u00e0 la d\u00e9tection d&#039;auto-anticorps coupables dans le s\u00e9rum.<span class=\"xref-bibr\">1<\/span> L&#039;IRM du cerveau a r\u00e9v\u00e9l\u00e9 des hyperintensit\u00e9s bilat\u00e9rales du claustrum sur l&#039;image pond\u00e9r\u00e9e en T2 (T2WI)\/fluid-attenuated inversion recovery (FLAIR) (figure 1). Auparavant, dans une cohorte de 34 patients,<span class=\"xref-bibr\">2<\/span> pr\u00e9sentant une \u00e9pilepsie r\u00e9fractaire d\u2019apparition r\u00e9cente, 4 patients sur 34 pr\u00e9sentaient des hypersignaux FLAIR de claustrum bilat\u00e9raux \u00e0 l\u2019IRM, aucun\u2026 <\/p>\n<p><span class=\"highwire-journal-article-marker-end\"><\/span><a href=\"http:\/\/pmj.bmj.com\/cgi\/content\/short\/98\/1166\/e24?rss=1\">Source quotidienne m\u00e9dicale<\/a><\/p>\n<p>","protected":false},"excerpt":{"rendered":"<p>Un jeune homme adulte s&#039;est pr\u00e9sent\u00e9 avec un d\u00e9but subaigu, une d\u00e9ficience cognitive rapidement progressive et des convulsions r\u00e9fractaires et a \u00e9t\u00e9 diagnostiqu\u00e9 comme une enc\u00e9phalite \u00e0 anticorps anti-r\u00e9cepteur N-m\u00e9thyl D-aspartate (anti-NMDAR) selon les crit\u00e8res de Graus <i>et coll.<\/i> (2016), suite \u00e0 la d\u00e9tection d&#039;auto-anticorps coupables dans le s\u00e9rum.<cross-ref type=\"bib\" refid=\"R1\">1<\/cross-ref> L&#039;IRM du cerveau a r\u00e9v\u00e9l\u00e9 des hyperintensit\u00e9s claustrales bilat\u00e9rales en image pond\u00e9r\u00e9e T2 (T2WI)\/r\u00e9cup\u00e9ration par inversion att\u00e9nu\u00e9e par fluide (FLAIR) (<cross-ref type=\"fig\" refid=\"F1\">Figure 1<\/cross-ref>). Auparavant, dans une cohorte de 34 patients,<cross-ref type=\"bib\" refid=\"R2\">2<\/cross-ref> pr\u00e9sentant une \u00e9pilepsie r\u00e9fractaire d&#039;apparition r\u00e9cente, 4 patients sur 34 pr\u00e9sentaient des hyperintensit\u00e9s bilat\u00e9rales du claustrum FLAIR \u00e0 l&#039;imagerie IRM, dont aucun n&#039;\u00e9tait positif pour les anticorps dirig\u00e9s contre les antig\u00e8nes onconeuraux. Tous ces patients souffraient d&#039;\u00e9pilepsies m\u00e9dicalement incurables chez lesquelles une imagerie r\u00e9p\u00e9t\u00e9e a r\u00e9v\u00e9l\u00e9 une r\u00e9solution des anomalies radiologiques, malgr\u00e9 des crises persistantes. Une atteinte radiologique du cortex bi-insulaire a d\u00e9j\u00e0 \u00e9t\u00e9 rapport\u00e9e dans le cadre d&#039;enc\u00e9phalites anti-NMDAR.<cross-ref type=\"bib\" refid=\"R3\">3<\/cross-ref> Cependant, dans notre cas, aucun changement insulaire n\u2019\u00e9tait perceptible ni aucune hyperintensit\u00e9 du claustrus. Le patient\u2026<\/p>","protected":false},"author":2,"featured_media":7458,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"Default","format":"standard","meta":{"page_builder":"","fifu_image_url":"https:\/\/urbancare.clinic\/wp-content\/uploads\/2023\/01\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis.jpg","fifu_image_alt":"","footnotes":""},"categories":[185],"tags":[],"class_list":["post-7457","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-research-medical-articles"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v28.4 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>Claustrum hyperintensity as a marker of anti-NMDAR encephalitis - Urban Care Clinic<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/urbancare.clinic\/fr\/hyperintensite-du-claustrum-comme-marqueur-de-lencephalite-anti-nmdar\/\" \/>\n<meta property=\"og:locale\" content=\"fr_FR\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Claustrum hyperintensity as a marker of anti-NMDAR encephalitis - Urban Care Clinic\" \/>\n<meta property=\"og:description\" content=\"A young adult male patient presented with subacute onset, rapidly progressive cognitive impairment and refractory seizures and was diagnosed as definite anti N-methyl D-aspartate receptor (anti-NMDAR) antibody encephalitis as per criteria by Graus et al. (2016), following detection of culprit autoantibodies in serum.1 MRI of brain revealed bilateral T2-weighted image (T2WI)\/fluid-attenuated inversion recovery (FLAIR) claustrum hyperintensities (figure 1). Previously, in a cohort of 34 patients,2 presenting with new-onset refractory epilepsy, 4 out of 34 patients showed bilateral claustrum FLAIR hyperintensities on MRI imaging, none of whom were positive for antibodies to onconeural antigens. All these patients had medically intractable epilepsies in whom repeat imaging revealed resolution of radiological abnormalities, despite ongoing seizures. Radiological involvement of bi-insular cortex has previously been reported in context of anti-NMDAR encephalitis.3 However, in our case, no insular changes were noticeable along with claustrum hyperintensities. 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