{"id":7457,"date":"2022-11-22T13:00:22","date_gmt":"2022-11-22T10:00:22","guid":{"rendered":"https:\/\/urbancare.clinic\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis\/"},"modified":"2022-11-22T13:00:22","modified_gmt":"2022-11-22T10:00:22","slug":"claustrum-hyperintensitat-als-marker-fur-anti-nmdar-enzephalitis","status":"publish","type":"post","link":"https:\/\/urbancare.clinic\/de\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis\/","title":{"rendered":"Claustrum-Hyperintensit\u00e4t als Marker f\u00fcr Anti-NMDAR-Enzephalitis"},"content":{"rendered":"<div><img decoding=\"async\" src=\"https:\/\/urbancare.clinic\/wp-content\/uploads\/2023\/01\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis.jpg\" class=\"ff-og-image-inserted\" style=\"display:none\"><\/div>\n<p><span class=\"highwire-journal-article-marker-start\"><\/span><\/p>\n<p id=\"p-1\">Ein junger erwachsener m\u00e4nnlicher Patient stellte sich mit subakutem Beginn, schnell fortschreitender kognitiver Beeintr\u00e4chtigung und refrakt\u00e4ren Anf\u00e4llen vor und wurde gem\u00e4\u00df den Graus-Kriterien eindeutig als Anti-N-Methyl-D-Aspartat-Rezeptor (Anti-NMDAR)-Antik\u00f6rper-Enzephalitis diagnostiziert <em>et al.<\/em> (2016), nach dem Nachweis der verantwortlichen Autoantik\u00f6rper im Serum.<span class=\"xref-bibr\">1<\/span> Die MRT des Gehirns zeigte bilaterale T2-gewichtete Bilder (T2WI)\/Fl\u00fcssigkeits-attenuierte Inversionswiederherstellung (FLAIR)-Claustrum-Hyperintensit\u00e4ten (Abbildung 1). Zuvor wurde in einer Kohorte von 34 Patienten<span class=\"xref-bibr\">2<\/span> 4 von 34 Patienten stellten sich mit neu aufgetretener refrakt\u00e4rer Epilepsie vor und zeigten bilaterale Claustrum-FLAIR-Hyperintensit\u00e4ten in der MRT-Bildgebung, keiner \u2026 <\/p>\n<p><span class=\"highwire-journal-article-marker-end\"><\/span><a href=\"http:\/\/pmj.bmj.com\/cgi\/content\/short\/98\/1166\/e24?rss=1\">Medizinische t\u00e4gliche Quelle<\/a><\/p>\n<p>","protected":false},"excerpt":{"rendered":"<p>Ein junger erwachsener m\u00e4nnlicher Patient stellte sich mit subakutem Beginn, schnell fortschreitender kognitiver Beeintr\u00e4chtigung und refrakt\u00e4ren Anf\u00e4llen vor und wurde gem\u00e4\u00df den Graus-Kriterien eindeutig als Anti-N-Methyl-D-Aspartat-Rezeptor (Anti-NMDAR)-Antik\u00f6rper-Enzephalitis diagnostiziert <i>et al.<\/i> (2016), nach dem Nachweis der verantwortlichen Autoantik\u00f6rper im Serum.<cross-ref type=\"bib\" refid=\"R1\">1<\/cross-ref> Die Magnetresonanztomographie des Gehirns ergab bilaterale T2-gewichtete Bilder (T2WI)\/Fluid-attenuated Inversion Recovery (FLAIR) Claustrum-Hyperintensit\u00e4ten (<cross-ref type=\"fig\" refid=\"F1\">Abbildung 1<\/cross-ref>). Zuvor wurde in einer Kohorte von 34 Patienten<cross-ref type=\"bib\" refid=\"R2\">2<\/cross-ref> Bei 4 von 34 Patienten mit neu aufgetretener refrakt\u00e4rer Epilepsie zeigten sich bilaterale Claustrum-FLAIR-Hyperintensit\u00e4ten in der MRT-Bildgebung, von denen keiner positiv auf Antik\u00f6rper gegen onkoneurale Antigene getestet wurde. Alle diese Patienten litten an medizinisch therapieresistenter Epilepsie, bei der wiederholte Bildgebung trotz anhaltender Anf\u00e4lle eine Aufl\u00f6sung radiologischer Anomalien zeigte. Eine radiologische Beteiligung des bi-insul\u00e4ren Kortex wurde bereits im Zusammenhang mit einer Anti-NMDAR-Enzephalitis berichtet.<cross-ref type=\"bib\" refid=\"R3\">3<\/cross-ref> In unserem Fall waren jedoch keine Inselver\u00e4nderungen und keine Claustrum-Hyperintensit\u00e4ten erkennbar. Der Patient\u2026<\/p>","protected":false},"author":2,"featured_media":7458,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"Default","format":"standard","meta":{"page_builder":"","fifu_image_url":"https:\/\/urbancare.clinic\/wp-content\/uploads\/2023\/01\/claustrum-hyperintensity-as-a-marker-of-anti-nmdar-encephalitis.jpg","fifu_image_alt":"","footnotes":""},"categories":[185],"tags":[],"class_list":["post-7457","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-research-medical-articles"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v28.4 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>Claustrum hyperintensity as a marker of anti-NMDAR encephalitis - Urban Care Clinic<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/urbancare.clinic\/de\/claustrum-hyperintensitat-als-marker-fur-anti-nmdar-enzephalitis\/\" \/>\n<meta property=\"og:locale\" content=\"de_DE\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Claustrum hyperintensity as a marker of anti-NMDAR encephalitis - Urban Care Clinic\" \/>\n<meta property=\"og:description\" content=\"A young adult male patient presented with subacute onset, rapidly progressive cognitive impairment and refractory seizures and was diagnosed as definite anti N-methyl D-aspartate receptor (anti-NMDAR) antibody encephalitis as per criteria by Graus et al. (2016), following detection of culprit autoantibodies in serum.1 MRI of brain revealed bilateral T2-weighted image (T2WI)\/fluid-attenuated inversion recovery (FLAIR) claustrum hyperintensities (figure 1). Previously, in a cohort of 34 patients,2 presenting with new-onset refractory epilepsy, 4 out of 34 patients showed bilateral claustrum FLAIR hyperintensities on MRI imaging, none of whom were positive for antibodies to onconeural antigens. All these patients had medically intractable epilepsies in whom repeat imaging revealed resolution of radiological abnormalities, despite ongoing seizures. Radiological involvement of bi-insular cortex has previously been reported in context of anti-NMDAR encephalitis.3 However, in our case, no insular changes were noticeable along with claustrum hyperintensities. 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